The endocrine system in chronic nitric oxide deficiency

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The endocrine system in chronic nitric oxide deficiency.

The experimental model of chronic inhibition of nitric oxide (NO) production has proven to be a useful tool to study cardiovascular and renal lesions produced by this type of hypertension, which are similar to those found in human hypertension. It also offers a unique opportunity to study the interaction of NO with the humoral systems, known to have a role in the normal physiology of vascular t...

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Nitric oxide deficiency in chronic kidney disease.

The overall production of nitric oxide (NO) is decreased in chronic kidney disease (CKD) which contributes to cardiovascular events and further progression of kidney damage. There are many likely causes of NO deficiency in CKD and the areas surveyed in this review are: 1. Limitations on substrate (l-Arginine) availability, probably due to impaired renal l-Arginine biosynthesis, decreased transp...

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Effect of nitric oxide deficiency on the pulmonary PTHrP system

Nitric oxide (NO) deficiency is common in pulmonary diseases, but its effect on pulmonary remodelling is still controversial. As pulmonary parathyroid hormone-related protein (PTHrP) expression is a key regulator of pulmonary fibrosis and development, the effect of chronic NO deficiency on the pulmonary PTHrP system and its relationship with oxidative stress was addressed. NO bioavailability in...

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Nitric oxide promotes distant organ protection: evidence for an endocrine role of nitric oxide.

Endothelial NOS (eNOS)-derived NO has long been considered a paracrine signaling molecule only capable of affecting nearby cells because of its short half-life in blood and relatively limited diffusion distance in tissues. To date, no studies have demonstrated that endogenously generated NO possesses a clearly defined endocrine function. Therefore, we evaluated whether enzymatic generation of N...

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Endocrine nitric oxide bioactivity and hypoxic vasodilation by inhaled nitric oxide.

Inhaled nitric oxide (iNO) is approved by the Food and Drug Administration for the treatment of persistent pulmonary hypertension of the newborn, largely as a result of the pioneering research efforts of Warren Zapol. Inhaled nitric oxide (NO) dilates pulmonary resistance vessels to improve ventilation–perfusion matching before being inactivated by reactions with hemoglobin (Hb) in blood exitin...

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ژورنال

عنوان ژورنال: European Journal of Endocrinology

سال: 2007

ISSN: 0804-4643,1479-683X

DOI: 10.1530/eje.1.02314